代表性成果
1、TFAM is an autophagy receptor that limits inflammation by binding to cytoplasmic mitochondrial DNA. Liu H, Zhen C, Xie J, Luo Z, Zeng L, Zhao G, Lu S, Zhuang H, Fan H, Li X, Liu Z, Lin S, Jiang H, Chen Y, Cheng J, Cao Z, Dai K, Shi J, Wang Z, Hu Y, Meng T, Zhou C, Han Z, Huang H, Zhou Q, He P, Feng D*. Nat Cell Biol. 2024 Jun;26(6):878-891.
2、Rpl12 is a conserved ribophagy receptor. Chen Y, Hu J, Zhao P, Fang J, Kuang Y, Liu Z, Dong S, Yao W, Ding Y, Wang X, Pan Y, Wu J, Zhao J, Yang J, Xu Z, Liu X, Zhang Y, Wu C, Zhang L, Fan M, Feng S, Hong Z, Yan Z, Xia H, Tang K, Yang B, Liu W, Sun Q, Mei K*, Zou W*, Huang Y*, Feng D*, Yi C*. Nat Cell Biol. 2025 Mar;27(3):477-492.
3、Oncogenic RAS induces a distinctive form of non-canonical autophagy mediated by the P38-ULK1-PI4KB axis. Wang X, Li S, Lin S, Han Y, Zhan T, Huang Z, Wang J, Li Y, Deng H, Zhang M*, Feng D*, Ge L*. Cell Res. 2025 Mar 7. doi: 10.1038/s41422-025-01085-9.
4、ATP9A Deficiency Causes ADHD and Aberrant Endosomal Recycling via Modulating RAB5 and RAB11 Activity. Meng T, Chen XT, He ZJ, Huang HF, Lin SY, Liu KR, Bai G, Xu MD, Zhuang HX, Zhang YL, Waqas A, Liu Q, Zhang C, Sun XD, Huang HS, Umair M*, Yan YS*, Feng D*. Molecular Psychiatry 2023 Jan 6;1-13. doi: 10.1038/s41380-022-01940-w.
5、Prohibitin 1 regulates mtDNA release and downstream inflammatory responses. Liu H, Fan H, He P, Zhuang H, Liu X, Chen M, Zhong W, Zhang Y, Zhen C, Li Y, Jiang H, Meng T, Xu Y, Zhao G, Feng D*. EMBO J. 2022 Oct 17;e111173. doi: 10.15252/embj.2022111173.
6、The AMPK-MFN2 axis regulates MAM dynamics and autophagy induced by energy stresses. Hu Y, Chen H, Zhang L, Lin X, Li X, Zhuang H, Fan H, Meng T, He Z, Huang H, Gong Q, Zhu D, Xu Y, He P*, Li L*, Feng D*. Autophagy. 2021 May;17(5):1142-1156.
7、Ca2+-triggered Atg11–Bmh1/2–Snf1 complex assembly initiates autophagy upon glucose starvation. Yao W, Chen Y, Zhang Y, Zhong S, Ye M, Chen Y, Fan S, Ye M, Yang H, Li Y, Wu C, Fan M, Feng S, He Z, Zhou L, Zhang L, Wang Y, Liu W, Tong J, Feng D*, Yi C*. J Cell Biol. 2024 Sep 2;223(9):e202310049. doi: 10.1083/jcb.202310049.